Presynaptic L-type Ca2+ channels increase glutamate release probability and excitatory strength in the hippocampus during chronic neuroinflammation

Download

Giansante G, Marte A, Romei A, Prestigio C, Onofri F, Benfenati F, Baldelli P, Valente P.

Journal of Neuroscience, 2020

Investigation of hyperexcitability in hippocampal circuits associated with neuroinflammation in epilepsy and other neurological disorder disorders.

Keywords: Epilepsy, Neuroinflammation, Mouse hippocampal neurons, Neurological disorders

Related Resources
In vitro astrocyte model to assess tetramethylenedisulfotetramine (TETS)-induced neuroinflammation
Tetramethylenedisulfotetramine alters Ca dynamics in cultured hippocampal neurons: mitigation by NMDA receptor blockade and GABA(A) receptor-positive modulation
Efficient generation of CA3 neurons from human pluripotent stem cells enables modeling of hippocampal connectivity in vitro

Related Products

Maestro Pro Product Image

Maestro Pro

Neural Software Module

Neural Module